শুক্রবার, ৩০ নভেম্বর, ২০১২

Oceanic crust breakthrough: Solving a magma mystery

Friday, November 30, 2012

Oceanic crust covers two-thirds of the Earth's solid surface, but scientists still don't entirely understand the process by which it is made. Analysis of more than 600 samples of oceanic crust by a team including Carnegie's Frances Jenner reveals a systemic pattern that alters long-held beliefs about how this process works, explaining a crucial step in understanding Earth's geological deep processes. Their work is published in Nature on November 29.

Magmas generated by melting of the Earth's mantle rise up below the oceanic crust and erupt on the Earth's surface at mid-ocean ridge systems, the longest mountain ranges in the world. When the magma cools it forms basalt, the planet's most-common rock and the basis for oceanic crust.

It has long been assumed that the composition of magmas erupting out of mid-ocean ridges is altered when minerals that form during cooling sink out of the remaining liquid, a process called fractional crystallization. In theory, trace elements that are not included in the crystallizing minerals should be little affected by this process, and their ratios should be the same in the erupting magma as they were in the original magma before cooling.

If this is true, trace element ratios in magmas erupting at mid-ocean ridges should represent those of the original parental magma that formed deep in the Earth's mantle. However, this process doesn't account for the high abundance of trace elements found in samples of basalt from mid-ocean ridges around the world, so the reality of the situation is obviously more complicated than previous theories indicated.

Using the extensive array of samples and advanced modeling, Jenner and her research partner Hugh O'Neill of the Australian National University demonstrated that the concentration of trace elements is due to the process by which the magma is cycled through the oceanic crust prior to being erupted on the sea floor at the mid-ocean ridges.

Magma collects under the Earth's surface in a pool of liquid rock called a magma chamber. Each chamber is frequently flushed with new magma, which mixes with the old magma that was already there, and then this blended magma erupts out onto the ocean floor. Following the influx of new magma and eruption, the remaining magma undergoes fractional crystallization. This means that minerals are separated out from the magma as it cools. However, these minerals contain only minor amounts of the trace elements. As a result, trace elements build up in the magma over time, as the magma chamber is continually replenished by new magma coming in to the system.

"It's a simple idea, but it fits remarkably well," Jenner said. "These new findings will permit us to explore the conditions of mantle melting and production of the Earth's most-common rock."

###

Carnegie Institution: http://www.ciw.edu

Thanks to Carnegie Institution for this article.

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Source: http://www.labspaces.net/125605/Oceanic_crust_breakthrough__Solving_a_magma_mystery

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বৃহস্পতিবার, ২৯ নভেম্বর, ২০১২

Cat Sickness - Pet Mania From The Pet Promenade Blog

Cats are one of the most popular pets in the United States. People love them for their interesting personality and beautiful fur. However, many people don?t know how to properly care for their pets or even know if they are sick. Like humans, cats also get sick. Of course, cats cannot directly communicate with us to let us know they need aid and attention. You need to be an observant pet owner in order to recognize the key signs of cat illness. You also need to know the basics of treating various cat diseases and ailments. If you are interested in learning more about caring for your cats, be sure to check out this article.

As a pet owner, you have to pay special attention to your furry friends. Minor changes in diet and behaviour can indicate sickness or unhappiness. This could be indicative of a small problem or a serious medical issue with your cat.

If you sense that your cat or pet is acting strangely, be sure to research their symptoms thoroughly. If you are unsure it is always a safe choice to visit a veterinarian near you.

One of the most obvious behavioural patterns that should catch your attention immediately is when your cat refuses to use the litter box. They might litter outside the house or in the house, but never touch a litter box. This can signify that your cat has kidney failure or a urinary tract infection. Both of these medical conditions are very serious and you should consulate your vet immediately if you suspect your cat has either of these problems.

Cat Sickness

Increased thirst and diet or decreased thirst and diet can also be an indicator on a troubling problem. If you notice your cat drinking a lot, they could possibly have pyometra or hyperthyroidism. On the other hand, loss of appetite could be caused by anaemia, injury, dehydration or stomach problems. Loss of appetite can also result if your cat has certain oral problems such as gingivitis or stomatitis. However if you own a kitty, you might expect to see an increase in their diet as they grow larger, so don?t worry too much.

A cat?s fur coat is also a great indicator for your cat?s happiness level. Cats love to lick and clean their fur as well as groom their bodies. If your cat has dirty or unattended fur, it could be a clue that your cat is stressed or doesn?t feel well. They could have malnutrition or be affected by parasites. They could also could have a skin disorder such as fleas. Both malnutrition and parasites can cause extreme harm to cats, especially if they are left untreated for a long period of time.

If you want to reduce your cat?s chance of sickness, be sure to give them plenty of attention and as well as the right quantity of food and water. Also, never hesitate to contact your vet if you suspect your cat is sick. I hope this article was helpful to you about cat sicknesses.

Resources To Aid You:

Cat Sickness
Cat Sickness
Cat Sickness

Source: http://www.petpromenadeblog.com/cat-sickness.html

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Study shows surge of bad disease genes in Europe

By Maggie Fox, NBC News

A scan of all the mutations in the human gene map shows something surprising ? people of European descent are evolving fast, and not for the better.

The study finds that in the past 5,000 years, European-Americans have developed a huge batch of potentially harmful genetic mutations ? many more than African-Americans.

The study, published in the journal Nature, may help explain why so many people develop diseases even though they don?t have common genetic mutations. It can also help explain why different people have so many different reactions to the same drug, said Joshua Akey of the University of Washington in Seattle who led the study.

It likely has to do with population explosion, Akey said. European populations expanded after the Ice Age ended and prosperous agricultural societies emerged. ?The number of mutations that exist is directly attributable to the population growth that happened in the last 5,000 years,? Akey told NBC News.

?The things that allowed us to go from millions to billions of has also been the same process that has been pumping in all these new mutations.?

Akey and colleagues at genetics institutions across the country examined the gene sequences of more than 6,500 people ? more than 4,200 European-Americans and 2,200 African-Americans. They were looking for small changes in the genetic code called single nucleotide variants ? one-letter differences in the genetic code of A,C, T and G.

They found ?an enormous excess of rare variants? in the European-Americans. And 73 percent of these mutations only appeared in the human genome in the past 5,000 to 10,000 years. Most were mutations that are known to weaken proteins, Akey said, and most of these harmful mutations were also in the people of European descent.

Now researchers are working to see which of these mutations might be associated with diseases. But many are in known disease-causing genes, such as the LAMC1 gene associated with premature ovarian failure, LRP1, which is linked with both Alzheimer?s disease and obesity and the CPE gene linked to hardening of the arteries.

Most are rare mutations ? meaning they only affect a few people. ?Some genes might be more disease-causing than other genes,? Akey said.

It may explain why it?s been so hard to find clear genetic links to many diseases. ?We have been looking for disease risk where it isn?t,? he said. ?The last five to 10 years have been dominated by looking for common genetic variations that dominate common diseases. There is a lot of disease risk that is unexplained. Maybe there are classes of mutations that haven?t been looked at.?

The findings could explain why some people can smoke for a lifetime and never get lung cancer or heart disease, while someone else might suffer a heart attack despite having healthy blood pressure and cholesterol levels.

It definitely shows evolution in action, Akey said. ?It?s just the process of evolution playing out in real time,? he said. ?The dramatic population expansions that occurred over the past couple thousand years had a profound consequence on our genetic variability.?

Genetic mutations usually occur by accident ? they are just mistakes that get made when DNA gets copied. They become important to evolution when they affect a person?s ability to survive and have children. The expansion of civilization, and the ability of societies to care for people who are less fit, was probably a factor in allowing these mutations to spring up, Akey said. ?I think that is undoubtedly true,? he said.

Some of the genes identified in the scan also affect peoples? response to drugs. That could explain why some people are helped, for example, by a cholesterol-lowering drug while others may not be. There wouldn?t have been much ?selective pressure? on these genes before the modern drug era, but that doesn?t mean the genes were not influenced by something else. ?It turns out that genes involved in adverse drug responses also have different biological roles,? Akey said ? for instance, detoxifying certain foods.

There may even be more evolution in the future, Akey predicted. One example ? phenylketonuria or PKU. It?s caused by a mutation in a gene that breaks down an amino acid called phenylalanine. People with PKU mutations must eat a strict, low-protein diet or they can develop seizures and mental retardation.

Now newborns are routinely tested for PKU so they can start the diet immediately and avoid any brain damage. Akey said because these kids can now grow up and lead normal lives, they will likely start having children and the gene may become more common in the population.

Related stories:

?

Source: http://vitals.nbcnews.com/_news/2012/11/28/15518402-study-shows-surge-of-bad-disease-genes-in-europeans?lite

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NSAID use linked to reduced hepatocellular carcinoma risk and mortality due to chronic liver disease

NSAID use linked to reduced hepatocellular carcinoma risk and mortality due to chronic liver disease [ Back to EurekAlert! ] Public release date: 28-Nov-2012
[ | E-mail | Share Share ]

Contact: Zachary Rathner
Zachary.Rathner@oup.com
301-841-1286
Journal of the National Cancer Institute

Researchers found that aspirin use is associated with a decreased risk of developing hepatocellular carcinoma and death from chronic liver disease (CLD), according to a study published November 28 in the Journal of the National Cancer Institute.

Hepatocellular carcinoma (HCC), the most common type of primary liver cancer, occurs mainly among patients with CLD. Previous reports have linked chronic inflammation due to CLD to cellular processes that could promote carcinogenesis. Because of their anti-inflammatory properties and widespread use to prevent cardiovascular and cerebrovascular disease, nonsteroidal anti-inflammatory drugs (NSAIDs) including aspirin and nonaspirin NSAIDs are being investigated as cancer chemopreventive agents. NSAIDs have been shown to have a beneficial effect in observational studies and clinical trials on risk of some cancers. However, the relationship between NSAID use and risk of HCC and death from CLD is unclear.

To investigate this relationship, Vikrant V. Sahasrabuddhe, M.B.B.S., Dr.P.H, from the Division of Cancer Epidemiology and Genetics at the National Cancer Institute, and colleagues, performed an observational study of 300,504 men and women aged 50 to 71 years enrolled in the National Institutes of Health-AARP Diet and Health Study who reported their aspirin and nonaspirin NSAID use and were followed-up for 10-12 years. The researchers linked the self-reported use of aspirin and nonaspirin NSAIDs to registry data on diagnoses of 250 cases of HCC and 428 deaths due to CLD to perform their study.

The researchers found that the use of NSAIDs was associated with a reduced risk of HCC and a reduced risk of death from CLD compared to non-users. Study participants who used aspirin had a 41% reduced risk of HCC and a 45% reduced risk of death from CLD, whereas those who used non-aspirin NSAIDs experienced a 26% reduced risk of CLD mortality but no reduced risk of HCC. The authors conclude that "these associations are prominent with the use of aspirin, and if confirmed, might open new vistas for chemoprevention of HCC and CLD."

In an accompanying editorial, Isra G. Levy, M.B., BCh., MSc., and Carolyn P. Pim, M.D., both from the Department of Epidemiology and Community Medicine at the University of Ottawa in Canada discuss how the known causes of chronic liver disease and primary liver cancer are hepatitis B and C virus infections, alcohol use, and a link between obesity and diabetes has been suggested. "We already have cheap, readily available interventions," such as vaccines for hepatitis B and C virus but "effective strategies for reduction of HBV and HCV are not always available or fully applied." Also, alcohol abuse and obesity are complex and multifactorial challenges that require interventions at the individual and system levels." They conclude that although we should study the potential of new chemopreventive strategies such as NSAID use, we should also continue to focus on improving the established practices and interventions.

###

Contact Info:

Article: NCI Office of Media Relations, ncipressofficers@mail.nih.gov

Editorial: Isra G. Levy, M.B., BCh., MSc., isra.levy@ottawa.ca


[ Back to EurekAlert! ] [ | E-mail | Share Share ]

?


AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.


NSAID use linked to reduced hepatocellular carcinoma risk and mortality due to chronic liver disease [ Back to EurekAlert! ] Public release date: 28-Nov-2012
[ | E-mail | Share Share ]

Contact: Zachary Rathner
Zachary.Rathner@oup.com
301-841-1286
Journal of the National Cancer Institute

Researchers found that aspirin use is associated with a decreased risk of developing hepatocellular carcinoma and death from chronic liver disease (CLD), according to a study published November 28 in the Journal of the National Cancer Institute.

Hepatocellular carcinoma (HCC), the most common type of primary liver cancer, occurs mainly among patients with CLD. Previous reports have linked chronic inflammation due to CLD to cellular processes that could promote carcinogenesis. Because of their anti-inflammatory properties and widespread use to prevent cardiovascular and cerebrovascular disease, nonsteroidal anti-inflammatory drugs (NSAIDs) including aspirin and nonaspirin NSAIDs are being investigated as cancer chemopreventive agents. NSAIDs have been shown to have a beneficial effect in observational studies and clinical trials on risk of some cancers. However, the relationship between NSAID use and risk of HCC and death from CLD is unclear.

To investigate this relationship, Vikrant V. Sahasrabuddhe, M.B.B.S., Dr.P.H, from the Division of Cancer Epidemiology and Genetics at the National Cancer Institute, and colleagues, performed an observational study of 300,504 men and women aged 50 to 71 years enrolled in the National Institutes of Health-AARP Diet and Health Study who reported their aspirin and nonaspirin NSAID use and were followed-up for 10-12 years. The researchers linked the self-reported use of aspirin and nonaspirin NSAIDs to registry data on diagnoses of 250 cases of HCC and 428 deaths due to CLD to perform their study.

The researchers found that the use of NSAIDs was associated with a reduced risk of HCC and a reduced risk of death from CLD compared to non-users. Study participants who used aspirin had a 41% reduced risk of HCC and a 45% reduced risk of death from CLD, whereas those who used non-aspirin NSAIDs experienced a 26% reduced risk of CLD mortality but no reduced risk of HCC. The authors conclude that "these associations are prominent with the use of aspirin, and if confirmed, might open new vistas for chemoprevention of HCC and CLD."

In an accompanying editorial, Isra G. Levy, M.B., BCh., MSc., and Carolyn P. Pim, M.D., both from the Department of Epidemiology and Community Medicine at the University of Ottawa in Canada discuss how the known causes of chronic liver disease and primary liver cancer are hepatitis B and C virus infections, alcohol use, and a link between obesity and diabetes has been suggested. "We already have cheap, readily available interventions," such as vaccines for hepatitis B and C virus but "effective strategies for reduction of HBV and HCV are not always available or fully applied." Also, alcohol abuse and obesity are complex and multifactorial challenges that require interventions at the individual and system levels." They conclude that although we should study the potential of new chemopreventive strategies such as NSAID use, we should also continue to focus on improving the established practices and interventions.

###

Contact Info:

Article: NCI Office of Media Relations, ncipressofficers@mail.nih.gov

Editorial: Isra G. Levy, M.B., BCh., MSc., isra.levy@ottawa.ca


[ Back to EurekAlert! ] [ | E-mail | Share Share ]

?


AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.


Source: http://www.eurekalert.org/pub_releases/2012-11/jotn-nul112612.php

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Studies examine whether therapies for heart failure are associated with improved survival

ScienceDaily (Nov. 27, 2012) ? An analysis of two heart failure therapies finds differing outcomes regarding improvement in survival, according to two studies appearing in the November 28 issue of JAMA.

In one study, Adrian F. Hernandez, M.D., M.H.S., of the Duke Clinical Research Institute, Durham, N.C., and colleagues examined the clinical effectiveness of aldosterone antagonist therapy and associations with long-term outcomes of older patients discharged from a hospitalization for heart failure.

"Aldosterone antagonist therapy [a diuretic drug] for heart failure and reduced ejection fraction [a measure of how well the left ventricle of the heart pumps with each contraction] has been highly efficacious in randomized trials. However, questions remain regarding the effectiveness and safety of the therapy in clinical practice," according to background information in the article.

The researchers examined outcomes of eligible patients hospitalized with heart failure and reduced ejection fraction using clinical registry data linked to Medicare claims from 2005 through 2010. The primary outcomes measured for the study were all-cause mortality, cardiovascular readmission, and heart failure readmission at 3 years, and readmission associated with hyperkalemia (higher than normal levels of potassium in the circulating blood) at 30 days and 1 year.

Of the 5,887 patients (average age, 78 years) who met the inclusion criteria from 246 hospitals, 1,070 (18.2 percent) received a prescription for an aldosterone antagonist at hospital discharge. The researchers found that rates of all-cause mortality (49.9 percent vs. 51.2 percent) and cardiovascular readmission (63.8 percent vs. 63.9 percent) were similar between the treatment groups at 3 years. The cumulative incidence rates of arrhythmia (5.4 percent vs. 3.9 percent) and elective readmission for an arrhythmia control device (6.5 percent vs. 4.2 percent) were higher for the treated group. Analysis of the data indicated that there were no significant differences in mortality and cardiovascular readmission.

The authors also found that the cumulative incidence of the first heart failure readmission was significantly lower in the treated group (38.7 percent vs. 44.9 percent). The hyperkalemia readmission rates at 30 days (2.9 percent vs. 1.2 percent) and 1 year (8.9 percent vs. 6.3 percent) were higher in the treated group; however, hyperkalemia was seldom the primary diagnosis for these readmissions, and the absolute increase in hyperkalemia as a primary diagnosis was small.

The authors add that a potential explanation for their findings is that aldosterone antagonists have limited effectiveness regarding mortality in real-world settings among older patients. "One potential reason for limited effectiveness may be a lack of adherence to or persistence with therapy. ? Our findings highlight the importance of conducting clinical trials that can be easily generalized to real-world practice and in which the most vulnerable patient groups are well represented."

Use of ACE Inhibitors, ARB's, Associated With Improved Survival Among Certain Patients With Heart Failure

In another study, Lars H. Lund, M.D., Ph.D., of the Karolinska Institutet, Stockholm, Sweden, and colleagues conducted a study to examine whether renin-angiotensin system (RAS) antagonists (i.e., angiotensin-converting enzyme [ACE] inhibitors or angiotensin receptor blockers [ARBs] are associated with reduced mortality in heart failure patients with preserved ejection fraction.

"Up to half of patients with heart failure have normal or near-normal ejection fraction, termed heart failure with preserved ejection fraction (HFPEF) or diastolic heart failure. The mortality in HFPEF may be as high as in heart failure with reduced ejection fraction (HFREF) or systolic heart failure, but there is no proven therapy," according to background information in the article.

The study included 41,791 patients in the Swedish Heart Failure Registry, from 64 hospitals and 84 outpatient clinics between 2000 and 2011. Of these, 16,216 patients with HFPEF (ejection fraction of 40 percent or greater; average age, 75 years; 46 percent women) were either treated (n = 12,543) or not treated (n = 3,673) with RAS antagonists. Analyses was conducted of the data to determine the association between use of RAS antagonists and all-cause mortality, with use of a matched cohort. The researchers included 20,111 patients with ejection fraction of less than 40 percent for the HFREF consistency analysis.

In the overall HFPEF group, crude 1-year survival was 86 percent for patients receiving RAS antagonists vs. 69 percent for patients not receiving RAS antagonists; and 5-year survival was 55 percent vs. 32 percent, respectively. In the matched HFPEF cohort, 1-year survival was 77 percent for treated patients vs. 72 percent for untreated patients. Five-year survival was 36 percent vs. 34 percent, respectively.

"There is currently no consensus on the use of RAS antagonists in patients with HFPEF. In our study, use of RAS antagonists was associated with reduced all-cause mortality in a broad unselected population of patients with HFPEF. Our results together with the signal toward benefit in randomized controlled trials suggest that RAS antagonists may be beneficial in patients with HFPEF, but this should be confirmed in an appropriately powered randomized trial," the authors conclude.

Editorial: Heart Failure Therapy -- What Should Clinicians Believe?

In an accompanying editorial, James C. Fang, M.D., of University Hospitals Case Medical Center, Cleveland, examines the question of what physicians should conclude from these 2 observational studies that would appear to contradict the clinical trial evidence.

"If all of the evidence is carefully considered in its totality, it would be sound to conclude that (1) renin-angiotensin system antagonists are reasonable agents to control hypertension in heart failure with preserved ejection fraction, and (2) aldosterone antagonists are effective drugs in heart failure with reduced ejection fraction but should be used carefully and selectively. Although clinical trials should remain the gold standard for testing hypotheses, observational studies bridge the gap from the scientific rigor of clinical trials to real-world experience. Clinical trials are a reminder of the rigor of medicine as a science; observational studies are a reminder that medicine is still an art."

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The above story is reprinted from materials provided by JAMA and Archives Journals.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.


Journal References:

  1. Hernandez AF, Mi X, Hammill BG, et al. Associations Between Aldosterone Antagonist Therapy and Risks of Mortality and Readmission Among Patients With Heart Failure and Reduced Ejection Fraction. JAMA: The Journal of the American Medical Association, 2012; 308 (20): 2097 DOI: 10.1001/jama.2012.14795
  2. Lund LH, Benson L, Dahlstr?m U, Edner M. Association Between Use of Renin-Angiotensin System Antagonists and Mortality in Patients With Heart Failure and Preserved Ejection Fraction. JAMA: The Journal of the American Medical Association, 2012; 308 (20): 2108 DOI: 10.1001/jama.2012.14785
  3. James C. Fang. Heart Failure Therapy: What Should Clinicians Believe? JAMA: The Journal of the American Medical Association, 2012; 308 (20): 2144 DOI: 10.1001/jama.2012.45418

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/top_news/top_health/~3/Qy1JbRtp7QY/121127190025.htm

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Cassini spots superstorm at Saturn's north pole

Lisa Grossman, physical sciences reporter

20121127_N00198348_filtered.jpg

(Image: NASA/JPL/Space Science Institute)

Hurricane Sandy?was a city-swamping superstorm, but the huge vortex churning at Saturn's north pole puts terrestrial storms to shame.

This image, captured on 27 November by NASA's Cassini spacecraft, stares into the storm's sinister eye from 361,000 kilometres away. The spacecraft observed in infrared wavelengths, which can peer through the top layer of clouds to reveal the complex texture beneath.

In 2007, the Cassini team saw a huge hexagon-shaped structure about 25,000 kilometres across stretching over Saturn's north pole. But the planet was in the depths of its 15-year-long winter, when sunlight does not fall on the pole, and it was too dark to see what lurked within the structure. Spring lifted the gloom in 2009, and now the team has spotted this vast storm at the hexagon's core.

Cassini scientists have seen something like this before. In 2006, the spacecraft observed Saturn's south pole, where a storm two-thirds as wide as Earth was raging. That vortex was the first place in the solar system other than Earth where astronomers saw eye-wall clouds, a typical feature of hurricanes in which a bank of clouds towers above the central pit.

Now, the springtime sun reveals a similar vortex swirling in the north. Astronomers think these storms form in the same way as hurricanes, with warm, moist air rising from lower cloud layers. The storms may be permanent, or could come and go with the seasons.

Read more about the sixth planet from the sun in our Saturn and its moons topic guide.

Subscribe to New Scientist Magazine

Source: http://feeds.newscientist.com/c/749/f/10897/s/260fa2e1/l/0L0Snewscientist0N0Cblogs0Cshortsharpscience0C20A120C110Csuperstorm0Esaturn0Bhtml0Dcmpid0FRSS0QNSNS0Q20A120EGLOBAL0Qonline0Enews/story01.htm

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Cassini spots superstorm at Saturn's north pole

Lisa Grossman, physical sciences reporter

20121127_N00198348_filtered.jpg

(Image: NASA/JPL/Space Science Institute)

Hurricane Sandy?was a city-swamping superstorm, but the huge vortex churning at Saturn's north pole puts terrestrial storms to shame.

This image, captured on 27 November by NASA's Cassini spacecraft, stares into the storm's sinister eye from 361,000 kilometres away. The spacecraft observed in infrared wavelengths, which can peer through the top layer of clouds to reveal the complex texture beneath.

In 2007, the Cassini team saw a huge hexagon-shaped structure about 25,000 kilometres across stretching over Saturn's north pole. But the planet was in the depths of its 15-year-long winter, when sunlight does not fall on the pole, and it was too dark to see what lurked within the structure. Spring lifted the gloom in 2009, and now the team has spotted this vast storm at the hexagon's core.

Cassini scientists have seen something like this before. In 2006, the spacecraft observed Saturn's south pole, where a storm two-thirds as wide as Earth was raging. That vortex was the first place in the solar system other than Earth where astronomers saw eye-wall clouds, a typical feature of hurricanes in which a bank of clouds towers above the central pit.

Now, the springtime sun reveals a similar vortex swirling in the north. Astronomers think these storms form in the same way as hurricanes, with warm, moist air rising from lower cloud layers. The storms may be permanent, or could come and go with the seasons.

Read more about the sixth planet from the sun in our Saturn and its moons topic guide.

Subscribe to New Scientist Magazine

Source: http://feeds.newscientist.com/c/749/f/10897/s/260fa2e1/l/0L0Snewscientist0N0Cblogs0Cshortsharpscience0C20A120C110Csuperstorm0Esaturn0Bhtml0Dcmpid0FRSS0QNSNS0Q20A120EGLOBAL0Qonline0Enews/story01.htm

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GOP divide surfaces early in 2014 Senate contest

WASHINGTON (AP) ? That didn't take long.

The fissures within the Republican Party that some say cost the GOP control of the Senate have resurfaced just three weeks after the election. This time conservatives are targeting a popular veteran congresswoman from a storied West Virginia political family making a bid for Democrat Jay Rockefeller's Senate seat in 2014.

Within an hour of Shelley Moore Capito's announcement of her candidacy, the influential and conservative Club for Growth branded her as the "establishment candidate" whose record in Congress of supporting prominent bailouts has led to bigger government. Capito just won her seventh term to Congress, securing about 70 percent of her district's vote. Her father, former Gov. Arch Moore, for years was the chief political rival of the man she hopes to replace in the Senate.

The new head of the Senate's GOP campaign arm, Sen. Jerry Moran of Kansas, dismissed the criticism from the right ? "I don't see this as damaging to her cause" ? but it's far from inconsequential in the Republicans' bid to retake the Senate.

Moran hasn't officially taken over as chairman of the National Republican Senatorial Committee yet, but he already finds himself defending a potential nominee who's widely popular in her state while trying to avoid alienating influential players on the party's right flank.

Downplaying the impact of the Club for Growth's criticism of Capito, Moran said Tuesday his committee hasn't made a decision on how heavily involved it will be in West Virginia's Republican Senate primary two years from now.

"Shelley Moore Capito is a known quantity in West Virginia," he said. "Her voting record is acceptable to the majority of West Virginians in her district for a long period of time. I don't see this as damaging to her cause."

Rockefeller, 75, has not said whether he'll seek a sixth term in the Senate, but Capito has the name recognition and fundraising ability to mount an effective campaign against an incumbent.

The Club for Growth wasted no time listing what it believes are her numerous faults. They likened her to candidates such as Rick Berg of North Dakota and Rep. Denny Rehberg of Montana, saying that while they were supposedly the most electable of the Republican candidates, they lost Senate races in Republican-leaning states.

"Her candidacy will undoubtedly be cheered by the GOP establishment, and dire warnings will be issued against any 'divisive' primary challenges, lest other candidates hurt Capito's chances of winning," said Club for Growth President Chris Chocola. "The problem is that Congresswoman Capito's record looks a whole lot like the establishment candidates who lost this year."

But many of the Club for Growth's candidates in recent elections also have stumbled badly. Richard Mourdock lost in Indiana after bouncing veteran Sen. Richard Lugar in the primary. In the 2010 elections, the organization threw its considerable financial backing behind Sharron Angle in Nevada and Ken Buck in Colorado, both losers to vulnerable Democratic incumbents.

Moran said the criticism of Capito from the Club for Growth was not unexpected.

"This is going to be decided not by the NRSC and not by the Club for Growth; it's going to be decided by the people of West Virginia," he said.

In the last election cycle, Democratic leaders in Washington didn't mind playing favorites during the primaries, heavily recruiting candidates they thought had the best chance of winning, while shunning some they did not see as formidable. GOP leaders, in contrast, sat back as their potential nominees fought it out.

Moran said figuring out the NRSC's role in the coming primaries will take a couple of months, and said his organization will play a role in some states.

"It's a state-by-state issue," he said.

Democratic officials, meanwhile, are enjoying the sideshow of a potential GOP split already in the works.

"Their argument is correct that the handpicked Republican establishment candidates did just as poorly as the more tea-party candidates," said Matt Canter, spokesman for the Democratic Senatorial Campaign Committee. "It's hard to argue with them in that respect."

Canter said the establishment candidates stumbled in part because of their efforts to appeal to tea party supporters and strong anti-government organizations such as the Club for Growth. He said an outsider might find it easy to defeat Capito, because GOP primaries in West Virginia typically do not attract a lot of voters.

"It would be very easy for a right-wing candidate to get the votes needed to win," Canter said.

Republican strategist Ron Bonjean said he would have preferred for the Club for Growth to have waited to see if a viable alternative to Capito emerges before attacking her. With Mitt Romney easily winning the state, Republicans figure to have a strong shot of winning the Senate seat in 2014.

"She's the best that state has to offer at this point," Bonjean said. "There's not a deep bench of Republican candidates who can immediately step into the fold, who can take on Sen. Rockefeller. Going after a female Republican right now when we lost the women's vote is not necessarily the wisest political move either."

Chocola said supporting fiscal conservatives such as Jeff Flake in Arizona and Ted Cruz in Texas, both Senate winners this month, is the best way back for the GOP.

"They are the future of the Republican Party," he said.

Source: http://news.yahoo.com/gop-divide-surfaces-early-2014-senate-contest-075553887--election.html

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2nd Saturn 'Pac-Man' moon revealed

New images from a NASA spacecraft orbiting Saturn have revealed a view any retro-gamer would love: a second moon with a heat tattoo of the 1980s video game icon Pac-Man.

The latest images were snapped by NASA's Cassini spacecraft during a photo session of Saturn's icy moon Tethys, revealing an infrared pattern on the moon shaped like Pac-Man. It is the second time Cassini has found a Pac-Man heat pattern on a Saturn moon using its infrared spectrometer. In 2010, the spacecraft found a similar view on Saturn's moon Mimas, which is also known for a giant impact crater that gives it a similar look to the fictional Death Star of "Star Wars" fame.

"Finding a second Pac-Man in the Saturn system tells us the processes creating these Pac-Men are more widespread than previously thought," study leader Carly Howett said in a statement Monday. "The Saturn system ? and even the Jupiter system ? could turn out to be a veritable arcade of these characters." [ Amazing Saturn Photos by Cassini ]

Scientists suspect that the Pac-Man shapes on Mimas and Tethys are created when high-energy electrons slam into low latitudes on the forward-facing sides of the moons as they orbit Saturn.

  1. Space news from NBCNews.com

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      Science editor Alan Boyle's blog: It looks as if someone is taking portraits of NASA's Curiosity rover on Mars from a few feet away ? but who's the photographer?

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This bombardment transforms the normally "fluffy" surface into hard-packed ice, NASA officials said. The effect means that the hard-packed ice does not heat up as fast during the day or cool down as fast at night, they added. ?

The surface of Tethys is also regularly bombarded by icy particles from geysers on Enceladus, another Saturn moon. The Pac-Man heat signature on Tethys, however, suggests that the surface changes from electron bombardment are occurring faster than the recoating effect from Enceladus' plumes, researchers said.

"Studies at infrared wavelengths give us a tremendous amount of information about the processes that shape planets and moons," Cassini spectrometer principal investigator Mike Flasar of NASA's Goddard Space Flight Center in Greenbelt, Md., explained. "A result like this underscores just how powerful these observations are."

Cassini's infrared spectrometer observations were obtained on Sept. 14, 2011. The research by Howett and her team is detailed in a recent edition of the science journal Icarus.

Howett and her colleagues found that the temperature on Tethys' surface varied depending on where in the Pac-Man they looked. The daytime temperature inside the Pac-Man shape's "mouth" was about 29 degrees Fahrenheit (minus 1.6 degrees Celsius) cooler than surrounding areas. The warmest temperature on Tethys, still a frigid minus 300 degrees F (minus 184 degrees C), was actually slightly colder than the warmest temperature on Mimas (about minus 290 degrees F or minus 178 degrees C).

Cassini's views of Tethys also confirmed that the Pac-Man heat map on the moon can also be spotted in visible-light images as a dark, lens-shaped area. The surface oddity was first sighted by NASA's Voyager spacecraft in 1980, but now finally explained.

"Finding a new Pac-Man demonstrates the diversity of processes at work in the Saturn system," said Cassini project scientist Linda Spilker of NASA's Jet Propulsion Laboratory in Pasadena, Calif. "Future Cassini observations may reveal other new phenomena that will surprise us and help us better understand the evolution of moons in the Saturn system and beyond."

NASA's Cassini spacecraft launched in 1997 and has been orbiting Saturn since its arrival at the ringed planet in 2004. Cassini is currently in the midst of an extended mission that runs through 2017.

Follow Space.com on Twitter @Spacedotcom. We're also on Facebook? and??Google+.

? 2012 Space.com. All rights reserved. More from Space.com.

Source: http://www.msnbc.msn.com/id/49982300/ns/technology_and_science-space/

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Urban Family Events: Nov 30th ? Dec 6th

2012 Festival of Trees?(Thursday, November 29th ? Sunday, December 2nd at the Shaw Conference Centre)?? Celebrate the start of the Christmas season by enjoying the festive displays of Christmas trees and other?d?cor?and attractions.

A Christmas Carol (Friday, November 30th ? Sunday, December 23rd at The Citadel Theatre) ? Take in this traditional Christmas classic showing for its 13th year!

A 21st Century Christmas Carol (Friday, November 30th at 7pm at the Jubilee Auditorium) ? Get your tickets for this family-friendly modern adaptation of the classic A?Christmas Carol.

Snowflake?Festival?Light Up?St. Albert (Friday, November 30th from 6:30pm ? 9pm in St. Albert) -?Celebrate the arrival of the Christmas season by participating in Christmas crafts, storytelling, visits with Santa and more!

African Penguin Visits (Saturday, December 1st from 1pm-3pm at West Edmonton Mall) ? Bring your kids to North Pole Place where they can visit Santa and some African Penguins!

Jingle On Indoor Santa Parade (Sunday, December 2nd from 10am-12pm in the downtown Edmonton pedway system) ? ?Enjoy a traditional Christmas parade while staying warm! This indoor parade features floats, marching bands, dancers and Santa himself.

WEM Playtime Presents Christmas Cookies at North Pole Place (Tuesday, December 4th from 10am-12pm at West Edmonton Mall) ? The first 100 kids to visit North Pole Place will get to help decorate the cookies that Santa?s elves have been busy baking.

Christmas Around The World (Tuesday, December 4th from 10am-11:30am at Muttart Conservatory) ? Register your little one for this program that allows them the opportunity to tour the pyramids, create a holiday craft, and learn about Christmas traditions around the world.

Christmas Reflections (Tuesday, December 4th & Thursday, December 6th from 6pm-10pm at Fort Edmonton Park) ? Enjoy the holiday season by touring Fort Edmonton Park in horse-drawn wagon then enjoy warm beverages, sing Christmas carols, eat Christmas treats and participate in even more fun activities.

The Walter?s Christmas Party (Wednesday, December 5th from 9:30am-11am at John Walter Museum) ? Register your 3-5 year old now for an old-fashioned Christmas party that allows children a chance to make traditional gifts, hear Christmas stories, learn about older traditions and take home special treats.

?Everyday Art? ? Tours For Tots?(Wednesday, December 5th?at the Art Gallery of Alberta)?- Bring your 3-5 year old and discover art in everyday things.

Legislature Light-up (Thursday, December 6th starting at 4pm at the Alberta Legislature) ? Take in the beautiful Christmas lights at the Legislature grounds while drinking hot chocolate and listening to choirs. The official light-up ceremony is on December 6th, but visit every evening until December 23rd and enjoy live music and free hot chocolate. Visit over the lunch hour and enjoy the live music then too!

Story Time with Mrs. Claus (Thursday, December 6th at 4pm at Sherwood Park Mall) ? Join Mrs. Claus for an hour of storytelling. The first 50 kids get a special treat too.

Star Wars Identities: The Exhibition?(Daily at Telus World of Science)?? Gather your?Star Wars?fans and be sure get your tickets to check out the?Star Wars Identities?exhibition now open until April!

If you?re?still?looking for things to do, check out some of our regular listings:

See a movie with your newborn/infant in a baby-friendly environment with the?Stars & Strollers?features (held in Edmonton on Wednesdays & Thursdays).?Also be sure to check out?Cineplex Family Favourites?where you can see favourite family movies (this week is?Home Alone) Saturday mornings for $2.50/ticket?at select theatres.

The?City of Edmonton?attractions offer many drop-in classes over the weekends. Be sure to check out the drop-in classes offered at:

As always, if you have a family-friendly event that should be featured on our Calendar, please send the details to?kjristen@urbaninfant.ca

Image:?Ted & Dani Percival?| Flickr.com

?

Source: http://www.urbaninfant.ca/2012/11/urban-family-events-nov-30th-dec-6th/?utm_source=rss&utm_medium=rss&utm_campaign=urban-family-events-nov-30th-dec-6th

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Fiction writing group seeks new members - Arts Talk - Capital ...

A 20-year-old writing workshop based in Albany is currently seeking new, dependable members with a passion for writing and critiquing and an ability to commit to twice-monthly, 90-minute meetings on Wednesday nights.

We?re an eclectic group of writers with day jobs, currently writing and reviewing novels, short stories and, occasionally, plays and screenplays. No poetry, please. We?re looking to expand our ranks with like-minded individuals who are as interested in talking about the written word as they are in putting it down on paper.

If interested, please tell us a bit about yourself and send a sample of writing to alwechs@juno.com.

Source: http://blog.timesunion.com/localarts/fiction-writing-group-seeks-new-members/25534/

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This week's sidebar poll: Are 1080p screens the next big thing?

DNA

We've seen a couple phones from HTC with a 1080p screen; namely the Droid DNA and J Butterfly, and have rumors of similar phones coming from Sony and LG. Clearly, someone in charge of the secret cabal of folks who designs smartphones likes the idea of a super high-resolution screen on a smartphone.

I just finished playing with a Droid DNA (knowing a mobile device reseller has its perks), and the screen looks awful damn nice. Because it's smaller in size, I say it looks as good (pixel wise) as the Nexus 10. But -- so does the HTC One X's 720p screen. In all honesty, I can't say the DNA screen is any better than the One X screen. Push the One X to 5-inches and maybe it's different, or maybe my eyes are tired from looking at Android devices for a few years. In either case, looking as good as the One X screen is nothing to be ashamed of.

Of course, the best part of the Android world is choice. So what I think really doesn't matter to anyone but me. 1080p screens are coming. Are the new must have? Tell us in the poll you'll find in the sidebar to the right or after the break.

Before we go, a look at last week's results.

Which has your interest -- the Galaxy Note 2 or the Droid DNA?

Poll

It's pretty conclusive that Samsung has things pretty well in hand for Verizon customers. We're not sure if one OEM having that much influence is a good or bad thing, but customer dollars do all the talking.

read more



Source: http://feedproxy.google.com/~r/androidcentral/~3/CriEJrpWaGY/story01.htm

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It's time to fix the charitable deduction - Fortune Finance: Hedge ...

By Mina Kimes

FORTUNE -- As elected officials in Washington struggle to find common ground on the deficit, it seems inevitable that tax breaks -- which, unlike tax rates, have been targeted by both parties -- will be on the chopping block. That includes the charitable deduction, which taxpayers can claim for donations to hospitals, colleges, churches, and other nonprofits. The Joint Committee on Taxation estimated that the tax break will cost the government $246 billion between 2010 and 2014.

The threat that lawmakers might eliminate -- or even curtail -- the charitable deduction has sent non-profits into a panic. The Charitable Giving Coalition, whose members include the United Way and the American Red Cross, recently announced plans to gather in the nation's capital on December 4th for a campaign called "Protect Giving - DC Days." The Independent Sector, a trade group for nonprofits, set up a website asking people to entreat their representatives to leave the deduction alone.

"We're seeing talk that we've never seen before, which suggests that we have a real issue here," says Diana Aviv, the head of the Independent Sector. Aviv says the tax break for donors should be protected because of its unique attributes. "The charitable deduction is not the same as other deductions," she says. "It doesn't benefit the individual."

Aviv is partially correct: the charitable tax break is different from many other tax breaks in so far as it clearly contributes to the public good. But the deduction does benefit individuals -- especially those in the upper class. According to a report by the Congressional Budget Office, taxpayers who make more than $100,000 a year took in 76% of the total charitable tax subsidy in 2006, despite contributing 57% of all donations. When wealthy people give money to charity, they reap outsized rewards.

Why the current deduction is unfair

There are several reasons for this discrepancy.?First, the tax break is a deduction, which means it can only be claimed by people who itemize their tax returns. That rules out the 70% of taxpayers who don't itemize. Second, because the expenditure is structured as a deduction, people in higher tax brackets can use it to net greater savings. Say a person in the 35% tax bracket donates $1000. If he or she deducts the contribution, his or her tax bill is reduced by 35% of $1000, or $350. Meanwhile, someone with a tax rate of 20% who donates the same amount of money will only save $200. As a result, it's cheaper for wealthy people to donate money.

MORE: Wall Street isn't backing Jack Lew for Treasury

By giving the rich a bigger incentive to donate, the government is effectively granting them greater control over the country's charitable giving. The subsidy is funded by all taxpayers, but the causes favored by the wealthy do not necessarily benefit everyone. PIMCO chief Bill Gross, himself a prominent philanthropist, told the New York Timesin 2007 that he thought wealthy donors were?over-compensated for giving money to "football stadiums and concert halls."?Gross added: "I don't think the public would vote for spending tax dollars on those things."

While lower-income taxpayers give 10% of their total donations to "basic needs organizations," according to the CBO, millionaires divert just 4% to such groups, preferring to donate to the arts and education sectors.?Some of those donations are used to pay for scholarships and charitable causes that benefit society at large, but other funds go to wealthy schools in high-income areas??In those cases, the government is essentially paying the rich to donate to their own communities.

Of course, many donations do go to worthy causes, none of which deserve to be starved of funding. But there's reason to believe that the charitable sector may be overstating the threat of a reduced tax break. Take, for example, the Charitable Giving Coalition's recent letter to President Obama, who proposed a couple of years ago that taxpayer deductions be limited to a rate of 28%. The Coalition argued that "any cap or limitation on charitable deductions" would undermine giving, with "long-lasting negative consequences." The Tax Policy Center has estimated that Obama's proposal would reduce private giving by about 2%.

That figure looks even smaller when you put it in the broader context of charities' revenue. In 2010, the nonprofit sector derived just 13% of its intake from private contributions. If you exclude hospitals and higher education organizations, which make most of their money from private payments and government sources, then the proportion of funding from private contributions increases to 24%.

Because the deduction has experienced little disruption since it was created in 1917, we cannot be absolutely sure what would happen if it were eliminated or cut. But there's reason to believe the effects would be smaller than previously thought. In recent years, several economists who have studied the price elasticity of giving, which is the percentage by which donations would decrease if the cost of giving were to go up, have found that the ratio is less than -1 -- meaning that, if the price went up by 1%, the level of giving would decline by less than 1%. A 2010 report by the Congressional Research Service points out that, historically, giving has not changed very much in response to changes in tax rates.

Many wealthy taxpayers say they would continue to donate if the deduction was reduced. In response to a recent survey conducted by the Center on Philanthropy at Indiana University, 50% of high-net-worth households said that they would give the same amount of money if the tax break were completely eliminated. "People tend to forget that some of the most significant giving in the U.S. dates back hundreds of years," says Rob Reich, an associate professor of political science at Stanford. "The Rockefellers and the Carnegies created foundations in the absence of any incentive whatsoever."

How to fix it: A floor and a credit

The charitable deduction is inequitable, costly, and inefficient. And yet, it should not be abolished altogether. For one, although economists have attempted to gauge the impact that eliminating the tax break would have on giving, the outcome is still uncertain; no one really knows what would happen (and which charities would suffer the most). Meanwhile, it's possible to reform the tax break and cut the subsidy while minimizing the impact on charitable giving.

Several politicians and think tanks have suggested that the tax break could be limited through the addition of a cap. An absolute dollar cap on deductions -- an idea promoted by Mitt Romney during his presidential campaign -- has been gaining steam. Such a proposal would effectively wipe out the charitable deduction, though,?because most people who itemize would first claim a deduction for their mortgage, which would consume most, if not all, of the allotted tax break.?President Obama's proposal for a 28% deduction cap -- described earlier -- would improve the structure of the tax break without hurting giving too badly, but it wouldn't raise very much money for the government.

MORE: Fiscal cliff: A modest proposal

A floor, which would force people to donate a certain amount of money to claim a tax break (and would exempt the money below the floor from the break, lowering the subsidy), offers a more elegant solution. The only people who would?who would donate less as a result of a floor would be those who contribute small amounts;?for others, there would be no reason to reduce giving at the margin. According to the Tax Policy Center, instituting a floor of 1.7% of adjusted gross income would raise $10-11 billion in annual revenue without affecting contributions at all. The CBO estimates that a floor of 2% of income would raise $15.7 billion while cutting donations by $3 billion.

The government could save even more money by converting the deduction into a tax credit, which would allow donors to claim a flat percentage of their donations. The CBO found that, if the charitable deduction were changed into a 25% credit with a floor of 2% of income, the government would cut the total subsidy by $11.9 billion a year, while donations would shrink by a mere $1 billion. A 15% credit would raise $24.6 billion, with donations falling by an estimated $10 billion, according to the CBO.

In the long-term, the savings would be significant. The Committee for a Responsible Federal Budget has estimated that changing the deduction to a 15% credit with a 2% floor would save the government $340 billion over the next decade, reducing the subsidy by 60%. Donations, meanwhile, would only decline by 4.9%. (The Bowles-Simpson commission proposed a similar, if slightly more draconian, 12% credit with a 2% floor.)

In addition to saving the government money, replacing the deduction with a credit would also make the system more equitable. All people would be equally compensated for giving to charity, regardless of their tax bracket. A credit would also reward the 70% of Americans who don't itemize their taxes, which might spur additional donations.

MORE: Wells Fargo CEO: Why Americans are saving so much

Such changes would inevitably change the profile of giving in this country, or at least the composition of donations that are subsidized by the government. People who make under $100,000 a year currently allot 67% of their donations to religious organizations, according to the CBO. Expanding the tax break to lower-income citizens would inevitably skew the subsidy toward churches.

This is a bad result. Not because it would compel taxpayers to fund widespread religious donations -- though many people would surely oppose such a large subsidy -- but because it would be wasteful. Studies have shown that the price elasticity of giving for religious donations is relatively low, which means that people would be unlikely to cut their gifts in response to a lowered tax break. Indeed, many religious donors do not currently claim a deduction.

If the government is serious about saving money, then it should consider exempting religious donations from the charitable tax break. Most people give to churches because they want to, not because they get a tax break for their generosity.

A massive subsidy would be not only controversial, but uneconomical.

Source: http://finance.fortune.cnn.com/2012/11/27/charitable-deduction-reform/

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AP PHOTOS: Scenes from Bangladesh factory fire

Firefighters battle a fire at a garment factory in the Savar neighborhood in Dhaka, Bangladesh, late Saturday, Nov. 24, 2012. At least 112 people were killed in a fire that raced through the multi-story garment factory just outside of Bangladesh's capital, an official said Sunday. (AP Photo/Hasan Raza)

Firefighters battle a fire at a garment factory in the Savar neighborhood in Dhaka, Bangladesh, late Saturday, Nov. 24, 2012. At least 112 people were killed in a fire that raced through the multi-story garment factory just outside of Bangladesh's capital, an official said Sunday. (AP Photo/Hasan Raza)

Civilians and firefighters battle a fire at a garment factory in the Savar neighborhood in Dhaka, Bangladesh, late Saturday, Nov. 24, 2012. An official said firefighters have recovered more than 100 bodies after a fire raced through the multi-story garment factory just outside Bangladesh's capital. (AP Photo/Polash Khan)

A firefighter douses the inside of a garment factory outside Dhaka, Bangladesh, Sunday, Nov. 25, 2012. At least 112 people were killed in a late Saturday night fire that raced through the multi-story garment factory just outside of Bangladesh's capital, an official said Sunday. (AP Photo/ khurshed Rinku)

A Bangladeshi woman cries as she claims the body of her relative killed in a fire at a garment factory outside Dhaka, Bangladesh, Sunday Nov. 25, 2012. At least 112 people were killed in a late Saturday night fire that raced through the multi-story garment factory just outside of Bangladesh's capital, an official said Sunday.(AP Photo/ Jibon Amir)

Bangladeshi police officers stand guard outside a burnt garment factory outside Dhaka, Bangladesh, Sunday, Nov. 25, 2012. At least 112 people were killed in a late Saturday night fire that raced through the multi-story garment factory just outside of Bangladesh's capital, an official said Sunday. (AP Photo/Khurshed Rinku)

A fire engulfed a garment factory outside Bangladesh's capital Dhaka, trapping many workers and killing at least 112 people in the building without emergency exits.

Authorities said the fire started on the ground floor late Saturday and spread upward, cutting off staircases and preventing workers' escape. Some survivors were rescued from the eight-story building's roof.

Thousands of onlookers and anxious relatives gathered at the burned-out factory Sunday awaiting word on relatives who worked there.

Bangladesh has some 4,000 garment factories, many without proper safety measures. The country annually earns about $20 billion from exports of garment products, mainly to the United States and Europe.

Here are photos of the fire and its aftermath:

Associated Press

Source: http://hosted2.ap.org/APDEFAULT/3d281c11a96b4ad082fe88aa0db04305/Article_2012-11-25-AS-Bangladesh-Fire-Photo-Gallery/id-a19b044c8ec9482698b0c562c507f704

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U.S. Planning A Force To Stay In Afghanistan

www.nytimes.com:

WASHINGTON ? American and allied military planners are drawing up the broad outlines of a force that would remain in Afghanistan following the handover to Afghan security after 2014, including a small counterterrorism force with an eye toward Al Qaeda, senior officials say.

Read the whole story at www.nytimes.com

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Source: http://www.huffingtonpost.com/2012/11/26/us-planning-a-force-to-st_n_2189788.html

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Activists: Syrian rebels seize major dam in north

BEIRUT (AP) ? Activists say Syrian rebels have captured a hydroelectric dam on the Euphrates river in the country's north in a strategic victory that followed days of fighting.

The Britain-based Syrian Observatory for Human Rights says the Tishrin Dam, near the town of Manbij, fell to the rebels before dawn on Monday.

Rami Abdul-Rahman, who heads the Observatory, says the dam supplies several areas of Syria with electricity.

The rebels have been making strategic advances recently. On Sunday, they briefly captured a regime helicopter base outside Damascus.

Syria's conflict started in March 2011 as uprising against President Bashar Assad's regime, inspired by other Arab Spring revolts.

It quickly morphed into a civil war that has since killed more than 40,000 people, according to activists.

Source: http://news.yahoo.com/activists-syrian-rebels-seize-major-dam-north-094500305.html

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Energy efficient windows ? Home Improvement: House Plans

November 20th, 2012 by admin Leave a reply ?

I must change the windows of my home. My old windows aren't bad looking. But although they go well with the house and I still like them, they are lacking the much needed energy efficiency. Heating and cooling my home takes five times longer than it is supposed to. I talked to the contractor that I always use, and after inspecting them, he told me that is time for windows upgrade. Because I really like the window design I have, I went in search for the similar window style only with a better energy efficiency index. I found something that looks promising at the Nitro Windows store my next door neighbor suggested. I?ll see soon enough how they will do.?

Source: http://redtreeinteractive.com/energy-efficient-windows/

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timothywell: How to prevent snoring? | Health and Fitness Tips ...

In everyday life, people who snore are not uncommon, many people think this is a performance sleeping, actually snore may not only lead to suffocation and even the night sudden death, and to influence others sleep.Today to tell you how to prevent snoring.

Methods / steps

1, Enhance physical exercise (preferably mountaineering, running, etc.), enhanced lung function.

2, Experts remind, the snoring if smoking habits you need to quit smoking immediately. Because smoking stimulus will only make the already clogged nasal and respiratory nasal mucosa worse. Drinking heavier snoring, nocturnal breathing disorders and hypoxemia. Especially bedtime Drinking.

3, Obese should actively lose weight.

4, Snoring patients more oxygen content decreased, often accompanied by high blood pressure, heart rhythm disorders, blood viscosity increased burden on the heart, easily lead to the occurrence of cardiovascular and cerebrovascular diseases, so attention should be paid to the monitoring of blood pressure, taking time to drop pressure drugs.

5, Bedtime prohibit taking sedative hypnotics and anti-allergic drugs, so as not to aggravate the conditioned inhibition of the respiratory center.

6, Take a lateral position sleep posture, especially in the right lateral position appropriate to avoid the tongue during sleep, the soft palate, uvula relaxation after the fall, adding to the blockage of the upper airway.

7, Patients after surgery to mainly soft food, do not eat hot food. Avoid strenuous activity.

8, Before going to bed try not to drink tea, coffee.

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Tags: harm of snoring, How to prevent snoring, how to stop snore, no snoring, prevent snoring, stop snore
This entry was posted on Friday, November 23rd, 2012 and is filed under Health Tips. You can follow any responses to this entry through the RSS 2.0 feed. You can leave a response, or trackback from your own site.

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LaCie Little Big Disk Thunderbolt (1TB SSD)


The LaCie Little Big Disk Thunderbolt (1TB SSD) ($999 list) is the latest version of the aluminum-clad thoroughbred series of external solid-state drives (SSDs), keeping a single user's data easily accessible at a moment's notice. With its Thunderbolt interface, it's one of the fastest drives we've ever tested, even rivaling a multi-mechanism RAID array costing twice as much. For the deadline-driven user that needed her job done yesterday, the Little Big Disk Thunderbolt (1TB SSD) is the Editor's Choice external drive to buy now.

Design and Features
Like the previous LaCie's Little Big Disk Thunderbolt (240GB SSD) ($899.99 list, 4 stars) , the 1TB SSD version of the Little Big Disk Thunderbolt is a pair of SSD drives installed into a 1.6-by-3.3-by-5.5-inch (HWD) chassis. All the drives in the Little Big Disk line (including the LaCie Little Big Disk Quadra ($450 list, 3.5 stars)) use the same chassis. The Little Big Disk chassis is constructed from aluminum, so the drive will survive the occasional drop in your commute bag. The sides are finned to help with passive cooling, so the tiny, single fan doesn't have to work too hard. That fan pumps out a bit of high-pitched noise, but thankfully the included Thunderbolt cable is long enough that you can put the drive behind your screen or into some other hidey hole. You can install the optional table stand for desktop use, but the drive works equally well without the stand. LaCie designed the Little Big Disk Thunderbolt to be a compact companion to Apple's line of MacBook Pro laptops, as well as Apple's desktop models.

The drive enclosure has two 512GB SATA III SSDs installed, giving you 1TB of combined storage space in RAID 0 (striping) configuration. The drive is set for RAID 0 out of the box. You can set the drive for RAID 1 (mirroring) using OS X's Disk Utility. RAID 1 will copy data to both SSDs simultaneously, giving you an instant backup at the expense of 500GB capacity. The included Thunderbolt cable (a $49 value) lets you plug the drive into any new Mac.

The drive has two Thunderbolt ports on the back (along with the jack for the AC adapter), so you can daisy chain several drives between your Mac and a Thunderbolt Display (or one with Mini DisplayPort). Unfortunately, like the other Little Big Disk drives, the 1TB SSD version is meant to be used on AC power: you can't power the drive solely from the Thunderbolt port, like the Editor's Choice for rugged portable drives, the LaCie Rugged USB 3.0 Thunderbolt (120GB SSD) ($299.99 list, 4.5 stars).

Since you can administer the RAID settings and erase the drive from OS X's Disk Utility, and since the drive is Time Machine compatible out of the box, the drive doesn't need any extra utilites. LaCie still includes a CD with LaCie Backup Assistant and the drive's documentation, plus you can download these files from www.lacie.com if you don't have an optical drive. The drive comes formatted for HFS+ (OS X native), but you can of course reformat the drive exFAT (an extension of FAT that supports 4GB+ files and is both Mac and PC compatible) or NTFS for PCs, provided your PC has a Thunderbolt port. The LaCie Little Big Disk Thunderbolt (1TB SSD) comes with a three-year warranty.

Performance
The LaCie Little Big Disk Thunderbolt (1TB SSD) is a speed demon. There, I've said it. The drive is capable of pushing 620MB/sec read, 380MB/sec write over the Thunderbolt cable, according to our AJA System Test. LaCie says it is capable of even faster performance with more drives daisy chained together. To put this into perspective, the Promise Pegasus R6 ($1,999 list, 3.5 stars) with six spinning hard drives costs twice as much, but manages a competitive 480MBps read, 620 MBps write score. This is astonishing, considering the price tags.

Generally, spinning drives are quicker to write to, but SSDs are quicker to read from. This is seen in the LaCie Rugged USB 3.0 Thunderbolt Series' Thunderbolt scores (379MBps read, 200MBps write using a single SSD) and the Western Digital My Book VelociRaptor Duo (Thunderbolt) (374MBps read, 343MBps write using two 10,000rpm spinning hard drives). The Little Big Disk Thunderbolt (1TB SSD) took only six seconds to copy our standard 1.2GB test folder, the same as the Pegasus R6. All the other drives took as much as three times as long under Thunderbolt.

If you're looking for a semi-portable, semi-rugged drive for your Thunderbolt-equipped Mac or PC, the LaCie Little Big Disk Thunderbolt (1TB SSD) should be at the top of the list (if you can afford it). Users who need to read multiple huge files (like HD videos) will want the Little Big Disk for its capacity and blazing read speeds. Since it's SSD-based storage, you won't have to worry about drive mechanisms wearing out, so it's a perfect drive to hold scientific or graphic libraries that your programs or websites need every second of every day. It'll also make a great work drive, holding both scratch files and the time-sensitive data files (media, graphics, video, database, etc.) you're working on right now. Our last Editors' Choice external hard drive, the IoSafe Solo G3 (1 TB) ($299 direct) won because of its innovative protection scheme and literally bullet-proof chassis. While we don't expect that the Little Big Disk will survive a bullet or fire, it is more rugged than plastic-clad drives, and the Little Big Disk Thunderbolt is one of the fastest drives we've ever tested. For users who need their job done minutes ago, the LaCie Little Big Disk Thunderbolt (1TB SSD) is the one you want.

COMPARISON TABLE
Compare the LaCie Little Big Disk Thunderbolt (1TB SSD) with several other hard drive side by side.

More hard drive reviews:
??? LaCie Little Big Disk Thunderbolt (1TB SSD)
??? Western Digital My Book Thunderbolt Duo
??? G-Technology G-Drive Mobile
??? Pioneer DVR-XD10
??? G-Technology G-Drive Slim
?? more

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